Dr Vladimir Zelenko, as part of a panel of COVID Experts, speaks to Australian Politicians, and explains how to stop virus replication: [254]
My involvement started at the epicentre of the largest outbreak in America, in the first week of March 2020. Thousands of patients needed treatment. They say “necessity is the mother of all innovation”. I had a big necessity to help my patients, keep them alive. I’m a family doctor for the last 20 years, for a community. So I had been in the game. Fortunately, with God’d help, I implemented an idea. That’s one of the advantages of being self-employed, and having a population of patients that trust you.
I observed improved breathing withing 6 to 12 hours. Initially I didn’t believe it, and then, after a few dozen patients I realised that there’s something here. My protocol was based on common sense; that’s my specialty. I wasn’t sure who was at risk. I called my colleagues at the local Intensive Care Units, and asked them as simple question: “which patients are dying?” They told me: “elderly patients, and, patients with medical problems”. I said, “what about the younger, healthier patients?” They told me they’ve not seeing any of them in ICU. So right away, I figured out that this virus doesn’t kill people.
Just to give you the sense of what’s going on, in my practice I used to see 50 patients, now we’re seeing between 200 and 300. There’ not enough resources for everyone. Half of my staff was sick. Services like radiology were unavailable, hospitals were at full capacity. So it was like a mass casualty event. And I had a triage, so I sent 2/3 of my patients home that I deemed at low risk, and focused my limited resources on the high-risk patients.
I didn’t wait to treat them, I used clinical judgement. I will do the testing, but at that time the testing took a week to get back, and I noticed that by the time the results came back the patients were half-dead, and that didn’t make sense to me, so I decided to treat them empirically, meaning, if I thought that they had COVID, I’m going to treat them for COVID.
I used data put together from Dr Didier Raoult work in Marcei, France, and from the South Koreans, based on a lecture from Dr Schultz from “Red Cream Episode 34”. He spoke about the use of Zinc, and Zinc dianophores; the concept being that Zinc inhibits viral replication.
Parenthetically: Most of the vaccine approach is about targeting the virus and preventing it from getting into the cell. Usually that happens by attacking the spike protein, which is the male part of the virus that attaches to the cell membrane, and the it gets absorbed into the cell. And all the variants that have clinical significance seem to have major mutations in the spike. Which basically means that the tri-dimensional shape of the spike protein has changed enough that perhaps existing anti-bodies that the patient may have from previous infections, or even from previous vaccination, may not recognise the new spike, hence be inefficient.
So my emphasis has never been un the prevention of the virus from getting into the cell. I don’t care about that. What I care is that, once the virus gets into the cell, it doesn’t make copies of itself.
There’s a mechanism, the pathway: the virus comes in, the genetic material contains in it the code for the protein fold, RNA-dependent RNA polymerase, and is very important because is used to make copies of the genetic material of the virus, which is then encapsulated, make a new virus, and then spread to other cells.
If we shut down that pathway, we shut down the ability of the virus to make copies of itself, then essentially, the immune system is capable of clearing the virus. The beauty of this pathway is that it’s a common pathway for all the variants. It doesn’t really matters to me if it is Omicron, or Delta, or whatever. What matters to me is that there’s another Zinc inside the cell, that when RNA-dependent RNA polymerase gets transcribed, that we could block it through a “monkey wrench” (sabotage) in our process, and hence stop the virus replication of all the variants of COVID19, and, again parenthetically, influenza virus. It just turned out that influenza virus also uses RNA-dependent RNA polymerase; and RSV virus, which supposedly has no treatment; and Marburg virus, which may be a potential future problem.
So, the whole key is to get the bullet into the cell, in enough concentration to block viral replication.
Well, there’s a problem with Zinc. It’s a charge, +2 ion, and dissolved in solution, it gets surrounded by water molecules, because H2O has a polarity, so essentially it gets covered by a water bubble around it. That’s a problem because the membrane is a lipid, think of it as cholesterol, like oil and water, and they don’t mix. So the Zinc works, but it doesn’t get into the cell. It’s like having a bullet without a gun.
So the idea is to use another substance to open a channel, a canal, in the cell membrane, and deliver the Zinc into the cell, in sufficient concentration, so that it can inhibit this virus, effectively. There’s a class of compounds that are known to do this:
So Hydroxicloroquine, and Ivermectin, and over-the-counter supplements like Quercetin, and something called ECGC which is a green tea extract. Under NHS server, there’re peer-reviewed papers on everything that I’m saying, if anyone is interested. The idea is, think of it as a gun, that delivers the Zinc, which is the bullet into the cell. Only the synergy of the two provides the functioning unit. So when you have the Zinc together with the Zinc ionophore, together you have a functioning “gun and bullet”.
Now the Zinc gets into the cell and blocks the RNA-dependent RNA polymerase. It doesn’t matter which variant, and how the virus got into the cell. We have a very efficient way of blocking RNA virus replication.
My empirical observations with the later dataset was published together with colleagues from Germany. Showed 84% reduction in hospitalisation. Statistically significant, very low P value. And then, subsequently, there’ve been dozens of other peer-reviewed studies that show earlier intervention with, it doesn’t matter which, anti-viral approach, there’re many now, but as long as you, in the high-risk patients, intervene in the right time frame, you can rid hospitalisation and death by 85%.
COVID-19 is two different disease processes:
There’s the viral phase, which no one dies from, it lasts around a week, and,
then, there’s the pathogenic catastrophic immune response, where the body’s immune system releases a cytokine disaster, which destroys human lung tissue, and causes blood clots. That happens after a week, or so, of having the virus.
So, conceptually, if we reduce the viral load, early in the disease process, then you’ve eliminated the complications, because they won’t happen. It’s more efficient to help a patient by getting rid of the virus early, than trying to deal with the complications in the lungs once they already happen. I think that’s common sense. It’s common sense in medicine that earlier intervention has better outcomes, my proof being that according to the CDC, the recommendation to treat influenza virus, which is an RNA virus, is to start anti-viral drugs within 48 hours. Why? Because that’s when they work.
Another example is cancer: It’s much better to treat cancer when it’s localised in one area, and not let it spread and become metastatic disease.